Anuradha Chakrabarty Email & Phone Number | My formal training was in biochemistry, but for last fourteen years I have been engaged in the studies of the nervous system. I have become fascinated by mechanisms of plasticity in the sensory nervous system, particularly with regard to pain pathways. I have explored the role of multiple mechanisms and molecules that lead to sensory nociceptor sprouting, a highly prevalent yet poorly studied phenomenon associated with several pain syndromes. My research has demonstrated the effect of estrogen on neurite outgrowth and sensory axon remodeling in inflammatory conditions. In a seminal study I reported that the angiotensin II receptor type 2 (AT2) expressed in sensory nociceptors is an essential regulator of axon sprouting, and estrogen acts on sensory neurons through upregulation of this receptor. These findings have opened new directions in our understanding of complex diseases like female pelvic pain syndromes and inflammatory pain. On the basis of this I have developed animal models in rat to study pain mechanisms in inflammation.

Anuradha Chakrabarty

My formal training was in biochemistry, but for last fourteen years I have been engaged in the studi...
2311 Marty AvenueApartment 5, Kansas City, United States

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Anuradha Chakrabarty’s Email achakrabarty@kumc.edu

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Anuradha Chakrabarty’s Location 2311 Marty AvenueApartment 5, Kansas City, United States

Anuradha Chakrabarty’s Current Industry University Of Kansas Medical Center

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University Of Kansas Medical Center

SENIOR SCIENTIST

2008-08-01T00:00:00.000Z - Present

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Anuradha Chakrabarty is located at
2311 Marty AvenueApartment 5, Kansas City, United States

Anuradha Chakrabarty is a My formal training was in biochemistry, but for last fourteen years I have been engaged in the studies of the nervous system. I have become fascinated by mechanisms of plasticity in the sensory nervous system, particularly with regard to pain pathways. I have explored the role of multiple mechanisms and molecules that lead to sensory nociceptor sprouting, a highly prevalent yet poorly studied phenomenon associated with several pain syndromes. My research has demonstrated the effect of estrogen on neurite outgrowth and sensory axon remodeling in inflammatory conditions. In a seminal study I reported that the angiotensin II receptor type 2 (AT2) expressed in sensory nociceptors is an essential regulator of axon sprouting, and estrogen acts on sensory neurons through upregulation of this receptor. These findings have opened new directions in our understanding of complex diseases like female pelvic pain syndromes and inflammatory pain. On the basis of this I have developed animal models in rat to study pain mechanisms in inflammation.